Blar i NTNU Open på forfatter "Aarset, Harald"

Viser treff 1-4 av 4

    • BRAF V600E mutation in early-stage multiple myeloma: good response to broad acting drugs and no relation to prognosis 

      Rustad, Even Holth; Dai, Hong Yan; Hov, Håkon; Coward, Eivind; Beisvag, Vidar; Myklebost, Ola; Hovig, Eivind; Nakken, Sigve; Vodak, Daniel; Meza-Zepeda, Leonardo A.; Sandvik, Arne Kristian; Wader, Karin Fahl; Misund, Kristine; Sundan, Anders; Aarset, Harald; Waage, Anders (Journal article; Peer reviewed, 2015)
      In this study, we analyzed the prevalence and clone size of BRAF V600E mutation in 209 patients with multiple myeloma and related the results to clinical phenotype, response and survival. Biopsies were screened for BRAF ...

    • In-hospital Cardiac Arrest Causes, Recognition and Survival 

      Bergum, Daniel (Doctoral theses at NTNU;2016:99, Doctoral thesis, 2016)
      Hjertestans på sykehus – En studie av underliggende årsaker. Tre studier ligger til grunn for denne avhandlingen. I disse studiene har vi gransket årsakene til hjertestans som oppstår plutselig og uventet inne på sykehus. ...

    • Symptomatic primary (Al) amyloidosis of the stomach and duodenum. 

      Fossmark, Reidar; Skarsvåg, Espen; Aarset, Harald; Hjort-Hansen, Henrik; Waldum, Helge (Journal article; Peer reviewed, 2013)
      Primary (AL) amyloidosis of the gastrointestinal tract is relatively rare, and symptomatic amyloidosis of the stomach is even more seldom. We present the case of a patient who was referred to upper endoscopy because of ...

    • The Norwegian PMS2 founder mutation c.989-1G>T shows high penetrance of microsatellite instable cancers with normal immunohistochemistry 

      Grindedal, Eli Marie; Aarset, Harald; Bjørnevoll, Inga; Røyset, Elin Synnøve; Mæhle, Lovise Olaug; Stormorken, Astrid T.; Heramb, Cecilie; Medvik, Heidi; Møller, Pål; Sjursen, Wenche (Journal article; Peer reviewed, 2014)
      Background Using immunohistochemistry (IHC) to select cases for mismatch repair (MMR) genetic testing, we failed to identify a large kindred with the deleterious PMS2 mutation c.989-1G > T. The purpose of the study was ...